Eating seaweed Ecklonia cava may have the option to decelerate or prevent Parkinson’s diseasein keeping with study 2024.
Scientists have discovered that antioxidants in Algae – often utilized in soups and salads in Asian cuisine – may protect our neurons from free radicals, stopping this devastating disease.
Parkinson’s disease is a disease that attacks the nervous system, causing symptoms comparable to tremors, stiffness, and difficulty moving. It happens when neurons that produce dopamine, a chemical that helps control movement, begin to die within the brain. Although Parkinson’s disease itself shouldn’t be fatal, it does cause serious complications that may result in death. Unfortunately, there isn’t any cure yet.
But scientists have already found links between dietary antioxidants and stopping Parkinson’s disease. For example, studies have shown that resveratrol – present in many plants and fruits, including red grapes, berries and peanuts – protected dopamine-producing neurons within the brain from death in several mouse models of Parkinson’s disease.
Ellagic acid, α-lipoic acid and myrtenal all improving the flexibility to learn and remember and neuromuscular coordination in mouse models of Parkinson’s disease. Ellagic acid present in many vegetables and fruit, including grapes, pomegranates, berries and nuts.
Myrtenal It is present in various plants comparable to hyssop and sage. Alpha lipoic acid is present in pork, spinach, and broccoli.
Several studies I suggested This drinking tea may help prevent Parkinson’s disease. Although we do not really understand how it really works, some research suggests that that is because of the presence of antioxidants within the tea.
Fighting free radicals
Our body produces harmful free radicals in response to environmental insults comparable to ultraviolet rays and air pollution. Free radicals are also natural by-product normal processes occurring in cells.
Antioxidants are like bodyguards, protecting cells from damage attributable to free radicals. Our bodies naturally produce antioxidants, but some foods, comparable to Ecklonia cava, are wealthy in antioxidants and might complement our naturally produced antioxidants help our bodies fight the harmful effects of free radicals.
For most last examinationbased in Japan, scientists induced Parkinson’s disease in mice using a pesticide rotenoneRotenone kills dopamine-producing neurons within the brain, causing mice to develop a Parkinson-like disease with similar symptoms, comparable to slower movements and impaired gastrointestinal motility (when there are problems with the movement of food and waste through the digestive tract).
When researchers induced Parkinson’s disease in mice, they fed a few of them antioxidants from Ecklonia cava and a few an everyday weight loss program. They found that dopamine-producing neurons within the brains of the mice fed the antioxidants appeared to be protected. In addition, the mice had fewer symptoms of Parkinson’s disease than the mice fed an everyday weight loss program.
The researchers also checked out the results of antioxidants in cells grown in a dish and exposed to rotenone. Rotenone increases the production of free radicals, killing cells.
Scientists have found that antioxidants reduce the production of free radicals induced by rotenone in cells, stopping cell death. This research opens up the potential for using Ecklonia cava polyphenols – plant compounds with antioxidant properties – to develop recent methods of treating and stopping Parkinson’s disease.
Lost in translation?
Unfortunately, the outcomes of animal and cell studies cannot at all times be extrapolated to humans. For example, despite protective effect Another antioxidant, vitamin C, which protects against Parkinson’s disease in cells and animal models, doesn’t appear to have the identical effect in humans.
This is because animal AND cell models don’t fully replicate Parkinson’s disease in humans. Animals have different brain structure and performance than humans, which implies that the way in which the disease develops and progresses could also be different. On the opposite hand, cellular models, even when scientists use human cells, lack of complexity.
Parkinson’s disease affects all the brain and body, involving many alternative cell types and interactions. Cellular models typically concentrate on a single cell type, missing this complexity.
Parkinson’s disease develops over a few years, symptoms change over time. Cellular and animal models cannot easily replicate this long-term progress. For example, the utmost lifespan of most laboratory mice is 2 years, while Parkinson’s disease is believed to develop for many years.
In conclusion, large-scale, robust clinical trials are needed to verify the effectiveness of Ecklonia cava in stopping or slowing down Parkinson’s disease. However, since Ecklonia cava is already available in dietary supplements, there may be probably no harm in taking it usually.
Will it prevent Parkinson’s? Maybe, but so will regular exercise.
